Conventional textbooks during this box have emphasised the fundamental sciences of pathology, biochemistry and body structure. Evidence-based Gastroenterology and Hepatology covers the entire significant ailments of the gastrointestinal tract and liver, using medical epidemiology to give the most powerful and most present proof for interventions.
This moment variation is edited and written by way of best gastroenterologists from worldwide, each one bankruptcy summarizes the proof in order that greater trained judgements should be made approximately which remedies to supply to patients.
It presents working towards Gastroenterologists and Surgeons with transparent information about the analysis and remedy of pancreatic illnesses, giving transparent proof and experience-based fabric that's instantly suitable to scientific practice.
Also incorporates a checklist of advised studying on the finish of every chapter.
Take a glance at brand new details at www.evidbasedgastro.com
Chapter 1 advent (pages 1–11): John WD McDonald, Brian G Feagan and Andrew okay Burroughs
Chapter 2 Gastroesophageal Reflux sickness (pages 13–54): Naoki Chiba
Chapter three Barrett's Esophagus (pages 55–68): Carlo A Fallone, Marc Bradette and Naoki Chiba
Chapter four Esophageal Motility issues: Achalasia and Spastic Motor problems (pages 69–81): Marcelo F Vela and Joel E Richter
Chapter five Ulcer sickness and Helicobacter Pylori (pages 83–116): Naoki Chiba
Chapter 6 Non?Steroidal Anti?Inflammatory Drug?Induced Gastroduodenal Toxicity (pages 117–138): Alaa Rostom, Andreas Maetzel, Peter Tugwell and George Wells
Chapter 7 Non?Variceal Gastrointestinal Hemorrhage (pages 139–159): Nicholas Church and Kelvin Palmer
Chapter eight practical Dyspepsia (pages 161–168): Sander JO Veldhuyzen van Zanten
Chapter nine Celiac ailment (pages 169–178): James Gregor and Diamond Sherin Alidina
Chapter 10 Crohn's illness (pages 179–195): Brian G Feagan and John WD McDonald
Chapter eleven Ulcerative Colitis (pages 197–210): Derek P Jewell and Lloyd R Sutherland
Chapter 12 Pouchitis After Restorative Proctocolectomy (pages 211–219): William J Sandborn
Chapter thirteen Microscopic and Collagenous Colitis (pages 221–229): Robert Lofberg
Chapter 14 Metabolic Bone affliction in Gastrointestinal problems (pages 231–246): Ann Cranney, Catherine Dube, Alaa Rostom, Peter Tugwell and George Wells
Chapter 15 Colorectal melanoma in Ulcerative Colitis: Surveillance (pages 247–253): Bret A Lashner and Alastair JM Watson
Chapter sixteen Colorectal melanoma: inhabitants Screening and Surveillance (pages 255–263): Bernard Levin
Chapter 17 Irritable Bowel Syndrome (pages 265–283): Albena Halpert and Douglas A Drossman
Chapter 18 Clostridium Difficile sickness (pages 285–301): Lynne V McFarland and Christina M Surawicz
Chapter 19 Ogilvie's Syndrome (pages 303–309): Michael D Saunders and Michael B Kimmey
Chapter 20 Gallstone disorder (pages 311–320): Calvin HL legislation, Dana McKay and Ved R Tandan
Chapter 21 Acute Pancreatitis (pages 321–339): Jonathon Springer and Hillary Steinhart
Chapter 22 weight problems (pages 341–357): Jarol Knowles
Chapter 23 Hepatitis C (pages 359–366): Patrick Marcellin
Chapter 24 Hepatitis B (pages 367–381): Piero Almasio, Calogero Camma, Vito Di Marco and Antonio Craxi
Chapter 25 Alcoholic Liver affliction (pages 383–391): Philippe Mathurin and Thierry Poynard
Chapter 26 Non?Alcoholic Fatty Liver sickness (pages 393–403): Chris P Day
Chapter 27 Hemochromatosis and Wilson sickness (pages 405–413): Gary Jeffrey and Paul C Adams
Chapter 28 basic Biliary Cirrhosis (pages 415–426): Jenny Heathcote
Chapter 29 Autoimmune Hepatitis (pages 427–434): Michael Peter Manns and Andreas Schuler
Chapter 30 basic Sclerosing Cholangitis (pages 435–451): Roger Chapman and Sue Cullen
Chapter 31 Portal Hypertensive Bleeding (pages 453–485): John Goulis and Andrew ok Burroughs
Chapter 32 Ascites, Hepatorenal Syndrome, and Spontaneous Bacterial Peritonitis (pages 487–503): Pere Gines, Vicente Arroyo and Juan Rodes
Chapter 33 Hepatic Encephalopathy (pages 505–515): Peter Ferenci and Christian Muller
Chapter 34 Hepatocellular Carcinoma (pages 517–525): Massimo Colombo
Chapter 35 Fulminant Hepatic Failure (pages 527–543): Nick Murphy and Julia Wendon
Chapter 36 Liver Transplantation: Prevention and therapy of Rejection (pages 545–571): Laura Cecilioni, Lucy Dagher and Andrew Burroughs
Chapter 37 Liver Transplantation: Prevention and remedy of an infection (pages 573–586): Nancy Rolando and Jim J Wade
Chapter 38 administration of Hepatitis B and C After Liver Transplantation (pages 587–601): George V Papatheodoridis and Rosangela Teixeira
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Additional info for Evidence-based Gastroenterology and Hepatology, Second Edition
These studies also support the notion that a symptom-based treatment is reasonable for most patients with reflux disease without a specific diagnosis. Treatment of gastroesophageal reflux disease Symptoms of gastroesophageal reflux are common and have a significant adverse impact on QOL. The costs of disease include both drug acquisition costs, and indirect costs such as physician visits and time off work. Because of the difficulty in make a definitive diagnosis of GERD through investigations, the physician must make a presumptive diagnosis and initiate a management plan.
The sensitivity and specificity of the sensor to predict esophageal acid exposure > 5% of time was 91% and 93%, respectively. This new probe is less expensive, disposable, easier to apply than the 24-hour pH probe and does not require recording equipment to be carried. Thus, there is the potential for this new method to greatly simplify esophageal pH measurements. 131 Erosive esophagitis (Savary–Miller grade 1 or worse) was identified in a third of patients with symptoms. Whether the patient had erosive disease or not, the frequency of heartburn and acid regurgitation correlated with median esophageal acid exposure time measured by 24-hour pH monitoring.
Abnormal score is placed 2 SD above the mean. This may not be valid as values do not follow a normal distribution 90% sensitivity and specificity Others have not reported the same results with the same criteria45 Schindlbeck et al. (1987)117 Upright time pH < 4 Supine time pH < 4 One or both above threshold = abnormal < 10·5% < 6·0% They assessed all the same factors as DeMeester above They carried out ROC analysis Klauser et al. (1989)16 Upright time pH < 4 Supine time pH < 4 One or both above threshold = abnormal < 8·2% < 3·0% Same group as Schindlbeck but larger reference sample gave lower threshold values Johnsson et al.