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Extra resources for Management of Morbid Obesity
Epilepsy Behav 2004; 5(1):88–93. 126. Cottam DR, Schaefer PA, Shatfan GW, Angus LD. Dysfunctional immune-privilege in morbid obesity: implications and effect of gastric bypass surgery. Obes Surg 2003; 13(1): 49–57. 127. Goodman E, Whitaker RC. A prospective study of the role of depression in the development and persistence of adolescent obesity. Pediatrics 2002; 110(3):497–504. 128. Sanchez-Roman S, Lopez-Alvarenga JC, Vargas-Martinez A, Tellez-Zenteno JF, VazquezVelazquez V, Arcila-Martinez D, Gonzalez-Barranco J, Herrera-Hernandez MF, SalinPascual RJ.
Left ventricular hypertrophy and heart failure. Independent of the arterial pressure, obesity increases the risk of left ventricular hypertrophy (LVH), especially the eccentric variant (44). Systolic function may remain intact in early obesity, but preload estimates suggest reduced contractility as measured by end-systolic wall stress and its relation to end–systolic volume index (45). Late systolic deterioration occurs through multiple mechanisms. Diastolic function also suffers, but these changes can be reversed by weight loss (46–48).
Similarities to intracranial hypertension would suggest a common etiology, however this has not been proven. Non-epileptic seizures. Psychogenic non-epileptic seizures (NES), physical manifestations of psychological distress, are associated with an increased risk of obesity. In a study by Marquez et al. 5 vs. 006) than controls, even when accounting for the potential weight gain associated with some anti-epileptic drugs. Whether this is related to the underlying psychiatric comorbidities of obesity, or is a separate phenomenon, is unclear (125).