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By J. V. Soames and J. C. Southam

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Rm1tans, but these could induce antibodies in humans which cross-react with heart tissue. Subsequently, various subunits of the organism have been investigated, especially surface antigens involved in the atrachment of the organism to tooth surfaces, which still confer protection against caries but without the risk of cross-reactivity. Immunization evokes a Immoral response and protection against S. m11tans is provided largely by secretory IgA antibodies in saliva, although IgG and IgM class antibodies can also gain access to the mouth via the crevicular fluid .

Dietary advice in dental practice. , 193, 563-7. Kidd, E. A. (1998). Assessment of caries risk. Dente!! te, 25, 385-90. ]. 0996). Dier and dental caries. ]. Murray), pp. 3- 31. Oxford University Press, Oxford. Kidd , E. A. M. (1996). The carious lesion in enamel. In The prevention rif dentC1! ]. Murray), pp. 95-106. Oxford University Press, Oxford. Kidd, E. A. , Ricketts, D. N. , and Pitts, N. B. (1993). Occlusal caries diagnosis: a changing challenge for clinicians and epidemiologists. ]ouma! of Dentistry, 21, 323-31.

Because of the strong evidence that S. 1m1tans and lactobacilli are major organisms associated with dental caries, simple screening rests to estimate the salivary levels of these bacteria have been developed as predictors of caries activity. However, the results from such tests need to be interpreted in conjunction with the assessment of other risk factors for caries in an individual patient. Infants become colonized by mutans streptococci from their mothers, and there is evidence that children of high-risk mothers become colonized at an earlier age and develop more carious lesions than children of low-risk mothers.

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